Seconder paroxysmal hemicrania caused by mucocele

Burc Esra Sahin, Ece Ozdemir Oktem

Abstract


Paroxysmal hemicrania (PH) is classified as a subgroup of trigeminal autonomic headaches among primary headaches. The pain is unilateral, located frequently at the orbital, supraorbital and/or temporal regions. It is sharp, severe, and these headaches tend to occur at least 5 times a day, which last for 2 – 30 minutes, accompanied by autonomic findings such as conjunctival injection, lacrimation, nasal congestion, nasal discharge, ptosis and orbital edema. Secondary PH is associated with tumor or vascular pathologies. Most frequently hypophyseal tumors were detected.In our case, there was a mucocele originating from the left ethmoidal sinus and compressing on the medial rectus muscle in the cranial MR examination of a patient admitted with clinical features of PH.In this case, it was seen that neuroimaging is mandatory in patients with TOB, in order to exclude secondary causes. Also, nociceptive impulses originating from surrounding tissues with the compression effect of the mucocele induce trigeminal autonomic reflex, supporting the hypothesis that autonomic symptoms and pain occur with hypothalamic and trigeminal connections.


Keywords


Neurology

Full Text:

PDF

References


Headache Classification Subcommittee of the International Headache Society. The International Classification of Headache Disorders, 3rd ed. Cephalalgia 33:629-808, 2013.

Indomethacin-responsive headaches. VanderPluym J. Curr Neurol Neurosci Rep 15:516, 2015.

Lambru G, Matharu MS. Trigeminal autonomic cephalalgias: A review of recent diagnostic, therapeutic and pathophysiological developments. Ann Indian Acad Neurol 15:51-61, 2012.

Fuad F, Jones NS. Paroxysmal hemicrania and cluster headache: two discrete entities or is there an overlap? Clin Otolaryngol Allied Sci. 27:472-479, 2002.

Prakash S, Belani P, Susvirkar A, Trivedi A, Ahuja S, Patel A. Paroxysmal hemicrania: a retrospective study of a consecutive series of 22 patients and a critical analysis of the diagnostic criteria. J Headache Pain 14:26, 2013.

Boes CJ, Dodick DW. Refining the clinical spectrum of chronic paroxysmal hemicrania: a review of 74 patients. Headache. 42:699-708, 2002.

Cittadini E, Matharu MS, Goadsby PJ. Paroxysmal hemicrania: A prospective clinical study of 31 cases. Brain. 131:1142-1155, 2008.

Sanjay Prakash, Rushad Patell. Paroxysmal Hemicrania: An Update Curr Pain Headache Rep 18:407, 2014.

Goadsy PJ, Lipton RB, A review of paroxismal hemicranias, SUNCT syndrome and other short- lasting headaches with autonomic feature, including new cases. Brain 120:193-209, 1997.

Prakash S, Hansen JM (2011) Mechanisms of cluster headache and other trigeminal autonomic

cephalalgias. In: Martelletti P, Timothy J, Steiner TJ (eds)

Handbook of headache: practical management, 1st ed. Springer Verlag, pp 330–340.

Matharu MS, Cohen AS, Frackowiak RSJ, Goadsby PJ. Posterior hypothalamic activation in paroxysmal hemicrania. Ann Neurol 59:535-545, 2006.

May A. Cluster headache: pathogenesis, diagnosis, and management. Lancet 366:843-855, 2005.

Malick A, Strassman RM, Burstein R. Trigemino hypothalamic and reticulohypothalamic tract neurons in the upper cervical spinal cord and caudal medulla of the rat. J Neurophysiol 84:2078-2112, 2000.


Refbacks

  • There are currently no refbacks.